Electromagnetic Therapy APPLICATION METHODOLOGY
The therapeutic effects of bio-electromagnetic therapy are far from always consistent, and many users of therapeutic devices like zapper or Rife generator question the reasons for fluctuating success rates. Why do some pathogens retreat with miraculous speed, while others, very ferocious, successfully resist electromagnetic attacks? We will therefore describe in more detail the biophysical mechanism of the effect of oscillating electromagnetic fields at the level of electro-sensitive biomolecules. This knowledge will allow us to better understand the issue and determine the correct strategy for the application of the pulsed EM field with regard to the considerable diversity of pathogenic microorganisms.
BASIC TYPES OF PATHOGENS
In terms of the mechanism of action, pathogens are divided in this way into 3 groups. Each of these three groups of pathogens requires a different method of application, duration of application and number of repetitions.
- RNA viruses (direct genome destruction)
- Bacteria with a short replication time (indirect genome destruction)
- Bacteria and DNA viruses with a long replication time (indirect genome destruction destruction) RNA VIRUSES The lower stability of RNA molecules makes them more sensitive to the effects of electromagnetic oscillating fields, which is why the method of applying resonance wavelengths of genomes in practice turns out to be the most effective for this group of viruses. Often, just one application of the correct frequency for about 15 minutes is sufficient to suppress the infection, However, some viruses possess mechanisms that repair or compensate for RNA damage. In addition, viruses can have multiple copies of their genetic material, allowing them to overcome a certain level of damage. For the stated reasons, in most cases it is advisable to repeat the frequency at least once in order to achieve the desired therapeutic effect. RNA viruses include, for example: Coronavirus including SARS-CoV-2, Influenza viruses, Enterovirus 72, Echoviruses, Hepatitis A, C, D, E, Poliovirus, Measles, Rotaviruses, Reoviruses, Respiratory syncytial virus, Rubella virus (rubella), Norovirus BACTERIA WITH A SHORT REPLICATION TIME The double helix structure of the DNA molecule contained in bacteria and DNA viruses is characterized by higher stability and resistance to external influences compared to the RNA molecule. DNA also has sophisticated repair mechanisms that minimize damage and mutations and help maintain its integrity. The application of resonant wavelengths can be effective even if the vibrations do not directly disintegrate the genome, as in the case of RNA viruses. During the application of resonant frequencies, mechanical vibration of the bacterial DNA occurs, which usually does not result in the direct damage to the genome, just as, for example, a microwave oven does not destroy the water molecule contained in food, it only vibrates it. It is important, however, what the consequences of such vibration of the genome are. This vibration of the genome leads to inactivation of repair mechanisms and the subsequent accumulation of genome disorders, which ultimately leads to self-destruction or apoptosis. In bacteria with a short replication period, the genome is damaged by the action of resonant frequencies in an indirect way. During the mechanical vibration of DNA, induced due to resonance frequencies, the genome is not only incapable of replication, but is not neither able to repair damage incurred during a failed attempt to replication. If we resonantly act with EM waves continuously for a corresponding period of time six times the reproductive period of a given bacterium, at each subsequent unsuccesfull replication attempt more and more errors and mutations acumulate until the level of damage to the genome becomes irreparable and the cell undergo programmed cell death (apoptosis). A typical example of a bacterium with a short replication (reproduction) time is E.coli, which doubles once every 40 minutes. The total therapy time should therefore last 40 minutes x 6 = 240 minutes. After this time, enough errors and damage should accumulate in the bacterium's genome. This time must be observed, because in the case of premature termination of therapy, only partial damage to the genome may occur, which will not be sufficient to stop the multiplication of bacteria. The bacteria will continue to replicate with the damaged genome, resulting in mutations that become resistant and no longer respond to the frequencies. A similar situation may arise in the case of intermittent use of antibiotics or their premature discontinuation. The time intervals below indicate the duration of one reproductive cycle and from that resulting total therapy time. Burkholderia cepacia: 1 hour (6 hours) Bacillus anthracis: 30 minutes (3 hours) Bacillus cereus: 20-30 minutes (2-3 hours) Bacteroides fragilis: 1 hour (6 hour) Bacterium lactis: 52 minutes (312 minutes) Bacillus subtilis: 20-30 minutes (120-180 minutes) Clostridium difficile: 50 minutes (300 minutes) Clostridium perfringens: 12 minutes (72 minutes) Enterococcus faecalis: 26 minutes (156 minutes) Enterococcus faecium: 40 minutes (240 minutes) Haemophilus influenzae: 30 minutes (180 minutes) Streptococcus pyogenes: 20-30 minutes (120-180 minutes) Lactobacillus acidophilus: 1 hours (6 hours) Listeria monocytogenes: 30-60 minutes (180-360 minutes) Legionella pneumohpila: 2 hours (12 hours) Micrococcus luteus: 1 hour (6 hour) Neisseria gonorrhoeae: 20-30 minutes (120-180 minutes) Neisseria meningitidis: 40 minutes (240 minutes) Proteus vulgaris: 28 minutes (168 minutes) Pseudomonas aeruginosa: 20-30 minutes (120-180 minutes) Salmonella enterica: 20-30 minutes (120-180 minutes) Salmonella typhimurium: 20 minutes (120 minutes) Serratia marcescens: 43 minutes (258 minutes) Shigella dysenteriae: 40 minut (240 minutes) Shigella flexneri: 40 minutes (240 minutes) Shigella sonnei: 32 minutes (192 minutes) Stenotrophomonas maltophilia: 30 minutes (180 minutes) Staphylococcus aureus: 30 minutes (180 minutes) Streptococcus agalactiae: 40 minutes (240 minutes) Streptococcus bovis: 27 minutes (162 minutes) Streptococcus haemolyticus A: 40 minutes (240 minutes) Streptococcus mitis: 40 minutes (240 minutes) Streptococcus mutans: 1 hour (6 hours) Streptococcus pneumoniae: 30-40 minutes (180-240 minutes) Streptococcus pyogenes: 40 minutes (240 min) Streptococcus salivarius: 30 minutes (180 minutes) Streptococcus sanguinus: 20 minutes (120 minutes) Yersinia enterocolitica: 34 minutes (204 minutes) BACTERIA WITH A LONG REPLICATION TIME Many species of bacteria and DNA viruses have very long replication times and therefore none of the strategies described can be applied. For example, spirochetes double their population within 30-50 hours, which is an incomparably longer time compared to bacteria with a short replication time. If we wanted to apply methodology of accumulation of chromosome damage by inactivation of repair mechanisms, described above, we would have to act unrealistically long. The goal of the new strategy targeting this large group of pathogens is not damage genome in a direct or indirect way, but to influence spatial organization, or conformational state of the entire DNA chromosome of the bacterium through the cell membrane. It is known that cellular membranes are receptors not only of chemical but also of electromagnetic signals. Electromagnetic waves interact with cell membranes, which act as amplifiers signal, which is then received by the DNA through the contact points where the DNA touches membranes. The primary targets of electromagnetic waves are the proteins involved in maintaining the structural and functional integrity of the DNA chromosome. Spatial organization of the chromosome can be influenced through the processes of molecular interaction under the influence of electromagnetic waves and associated changes in the secondary structure of DNA lead to disruption of transcription processes, reparation and especially replication.
- Bacteria with a short replication time (indirect genome destruction)
- Bacteria and DNA viruses with a long replication time (indirect genome destruction destruction) RNA VIRUSES The lower stability of RNA molecules makes them more sensitive to the effects of electromagnetic oscillating fields, which is why the method of applying resonance wavelengths of genomes in practice turns out to be the most effective for this group of viruses. Often, just one application of the correct frequency for about 15 minutes is sufficient to suppress the infection, However, some viruses possess mechanisms that repair or compensate for RNA damage. In addition, viruses can have multiple copies of their genetic material, allowing them to overcome a certain level of damage. For the stated reasons, in most cases it is advisable to repeat the frequency at least once in order to achieve the desired therapeutic effect. RNA viruses include, for example: Coronavirus including SARS-CoV-2, Influenza viruses, Enterovirus 72, Echoviruses, Hepatitis A, C, D, E, Poliovirus, Measles, Rotaviruses, Reoviruses, Respiratory syncytial virus, Rubella virus (rubella), Norovirus BACTERIA WITH A SHORT REPLICATION TIME The double helix structure of the DNA molecule contained in bacteria and DNA viruses is characterized by higher stability and resistance to external influences compared to the RNA molecule. DNA also has sophisticated repair mechanisms that minimize damage and mutations and help maintain its integrity. The application of resonant wavelengths can be effective even if the vibrations do not directly disintegrate the genome, as in the case of RNA viruses. During the application of resonant frequencies, mechanical vibration of the bacterial DNA occurs, which usually does not result in the direct damage to the genome, just as, for example, a microwave oven does not destroy the water molecule contained in food, it only vibrates it. It is important, however, what the consequences of such vibration of the genome are. This vibration of the genome leads to inactivation of repair mechanisms and the subsequent accumulation of genome disorders, which ultimately leads to self-destruction or apoptosis. In bacteria with a short replication period, the genome is damaged by the action of resonant frequencies in an indirect way. During the mechanical vibration of DNA, induced due to resonance frequencies, the genome is not only incapable of replication, but is not neither able to repair damage incurred during a failed attempt to replication. If we resonantly act with EM waves continuously for a corresponding period of time six times the reproductive period of a given bacterium, at each subsequent unsuccesfull replication attempt more and more errors and mutations acumulate until the level of damage to the genome becomes irreparable and the cell undergo programmed cell death (apoptosis). A typical example of a bacterium with a short replication (reproduction) time is E.coli, which doubles once every 40 minutes. The total therapy time should therefore last 40 minutes x 6 = 240 minutes. After this time, enough errors and damage should accumulate in the bacterium's genome. This time must be observed, because in the case of premature termination of therapy, only partial damage to the genome may occur, which will not be sufficient to stop the multiplication of bacteria. The bacteria will continue to replicate with the damaged genome, resulting in mutations that become resistant and no longer respond to the frequencies. A similar situation may arise in the case of intermittent use of antibiotics or their premature discontinuation. The time intervals below indicate the duration of one reproductive cycle and from that resulting total therapy time. Burkholderia cepacia: 1 hour (6 hours) Bacillus anthracis: 30 minutes (3 hours) Bacillus cereus: 20-30 minutes (2-3 hours) Bacteroides fragilis: 1 hour (6 hour) Bacterium lactis: 52 minutes (312 minutes) Bacillus subtilis: 20-30 minutes (120-180 minutes) Clostridium difficile: 50 minutes (300 minutes) Clostridium perfringens: 12 minutes (72 minutes) Enterococcus faecalis: 26 minutes (156 minutes) Enterococcus faecium: 40 minutes (240 minutes) Haemophilus influenzae: 30 minutes (180 minutes) Streptococcus pyogenes: 20-30 minutes (120-180 minutes) Lactobacillus acidophilus: 1 hours (6 hours) Listeria monocytogenes: 30-60 minutes (180-360 minutes) Legionella pneumohpila: 2 hours (12 hours) Micrococcus luteus: 1 hour (6 hour) Neisseria gonorrhoeae: 20-30 minutes (120-180 minutes) Neisseria meningitidis: 40 minutes (240 minutes) Proteus vulgaris: 28 minutes (168 minutes) Pseudomonas aeruginosa: 20-30 minutes (120-180 minutes) Salmonella enterica: 20-30 minutes (120-180 minutes) Salmonella typhimurium: 20 minutes (120 minutes) Serratia marcescens: 43 minutes (258 minutes) Shigella dysenteriae: 40 minut (240 minutes) Shigella flexneri: 40 minutes (240 minutes) Shigella sonnei: 32 minutes (192 minutes) Stenotrophomonas maltophilia: 30 minutes (180 minutes) Staphylococcus aureus: 30 minutes (180 minutes) Streptococcus agalactiae: 40 minutes (240 minutes) Streptococcus bovis: 27 minutes (162 minutes) Streptococcus haemolyticus A: 40 minutes (240 minutes) Streptococcus mitis: 40 minutes (240 minutes) Streptococcus mutans: 1 hour (6 hours) Streptococcus pneumoniae: 30-40 minutes (180-240 minutes) Streptococcus pyogenes: 40 minutes (240 min) Streptococcus salivarius: 30 minutes (180 minutes) Streptococcus sanguinus: 20 minutes (120 minutes) Yersinia enterocolitica: 34 minutes (204 minutes) BACTERIA WITH A LONG REPLICATION TIME Many species of bacteria and DNA viruses have very long replication times and therefore none of the strategies described can be applied. For example, spirochetes double their population within 30-50 hours, which is an incomparably longer time compared to bacteria with a short replication time. If we wanted to apply methodology of accumulation of chromosome damage by inactivation of repair mechanisms, described above, we would have to act unrealistically long. The goal of the new strategy targeting this large group of pathogens is not damage genome in a direct or indirect way, but to influence spatial organization, or conformational state of the entire DNA chromosome of the bacterium through the cell membrane. It is known that cellular membranes are receptors not only of chemical but also of electromagnetic signals. Electromagnetic waves interact with cell membranes, which act as amplifiers signal, which is then received by the DNA through the contact points where the DNA touches membranes. The primary targets of electromagnetic waves are the proteins involved in maintaining the structural and functional integrity of the DNA chromosome. Spatial organization of the chromosome can be influenced through the processes of molecular interaction under the influence of electromagnetic waves and associated changes in the secondary structure of DNA lead to disruption of transcription processes, reparation and especially replication.