Custom Frequencies
ALS
Enteroviruses, including poliovirus, coxsackievirus, echovirus, enterovirus-A71, and enterovirus-D68, are thought to contribute to the development of ALS because they can target motor neurons.
Endogenous retroviruses are also likely to be involved in the pathophysiology of ALS. Antibodies against this HERV-K virus have been described in 25-45% of ALS patients. ALS risk is also associated with human herpesvirus-6 and herpesvirus-8 seropositivity.
Alzheimer disease
It becomes more and more apparent that the AD pathology might originate from brain infection.
There is no unique Alzheimer or dementia germ,” instead, it is a general reaction of the CNS to chronic infections, in the environment of an aged immune system. Two protocols are focused on most guilty pathogens. However, the pathology may become self-sustained even without continuous presence of microbes in the brain, so the success is not guaranteed.
There is no unique Alzheimer or dementia germ,” instead, it is a general reaction of the CNS to chronic infections, in the environment of an aged immune system. Two protocols are focused on most guilty pathogens. However, the pathology may become self-sustained even without continuous presence of microbes in the brain, so the success is not guaranteed.
Antiphospholipid Syndrome
High levels of IgM antibodies against Rubella, Toxoplasma gondii, Cytomegalovirus (CMV), and hepatitis C virus have been found. Moreover, cross-reactivity has been demonstrated between proteins of bacteria such as Haemophilus influenzae, Neisseria gonorrhoeae, tetanus toxoid, and CMV with anti β2 glycoprotein 1 (β2-GPI) antibodies, which is one of the typical antibodies of this syndrome.
Basalioma
The cause of skin tumors, including basal cell carcinoma, can be a disruption of the balance of the skin microbiome, especially the suppression of beneficial bacteria (Staphylococcus epidermidis and Cutinobacterium), leading to an overgrowth of the Staphylococcus aureus bacterium, which is usually positive in biopsies performed in patients with basal cell carcinoma. Suppression of Staphylococcus should help restore the balance, which can significantly contribute to solving the problem, because S. epidermidis in particular has an important protective function - it prevents the further spread of tumor cells. It is therefore very important to eliminate pathogenic S. aureus and to achieve the re-proliferation of S. epidermidis.
Dementia
This protocol is composed of frequencies targeted on pathogens found in dementia patients brains post-mortem, such as various dental infections, herpes viruses, etc. There are 46 frequencies and 4 sweeps, that should be divided into 3 - 4 programs at least.
Guillain-Barre Syndrome
The protocol consists of frequencies focused on most common causes of this disease - campylobacter jejuni, cmv, ebv, zoster, chlamydia pneu. The protocol includes Nogier frequencies to support nerves regeneration.
Multiple sclerosis
There are different types of viruses and bacteria associated with the induction of autoantibodies to myelin basic protein (e.g., Acinetobacter sp. and P. aeruginosa). These infections are associated with the occurrence of autoantibodies against MBP and myelin glycoprotein oligodendrocyte due to their similarity with some bacterial molecules. Another infection of EBV causes the onset of MS partially due the disease’s symptoms being undetected during the earliest stages and partially due to the evolving relationship between EBV and the host’s immune system, which is repeatedly stimulated whenever latent virus reactivates.
Rheumatoid arthritis
It has been reported that the IgM response to some bacterial infections, e.g., Escherichia coli, Klebsiella pneumonia, and Proteus mirabilis is associated with rheumatoid factor. In the case of P. Miriabilis, a relationship with RA was established through structural similarities between self-epitopes and bacterial molecules. Additionally, RA has also been associated with the presence of Hepatitis B virus which is higher in RA patients than in healthy controls. Similar findings have been reported for EBV in RA as well as for Sjögren’s syndrome.
Systemic lupus erythematosus
Studies of SLE ona animals have shown that Epstein-Barr virus (EBV) can trigger the production of autoreactive antibodies with subsequent development of manifestations similar to those presented in the human disease. In patients, a high EBV seroprevalence has been observed as compared to healthy controls. Additionally, it has been suggested that Rubella and CMV may induce the production of autoantibodies in patients with SLE. Toxoplasma gondii and Helicobacter pylori are also risk factors for autoimmunity,